Vertiginous epilepsies are included in the category of the partial epilepsy in which abnormal electrical activity in the brain is localized.
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On November 30, 1999, levetiracetam was approved for the adjunctive treatment of partial epilepsy in adults in the United States.
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Symptomatic women with subependymal heterotopia typically present with partial epilepsy during the second decade of life; development and neurologic examinations up to that point are typically normal.
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An inherited form of human epilepsy known as autosomal dominant partial epilepsy with auditory features ( ADPEAF ) has been found to be caused by mutations of the LGI1 gene.
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Following genetic linkage studies placing the hereditary form of autosomal dominant partial epilepsy with Auditory features ( ADPEAF ) on chromosome region 10q24 mutation analysis of affected members in these families demonstrated LGI1 was the cause of the disease.
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In 1990, primidone, along with phenobarbital, was a second-line agent in partial epilepsy with or without secondarily generalized tonic-clonic seizures and was one of four agents ( the others being carbamazepine, phenytoin and phenobarbital ) that was used along with ethosuximide or a benzodiazepine for any myoclonic seizures when valproate failed to control tonic-clonics ( at least in the United States ).
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